Gout is not only against joint
People generally believe that the main cause gout joint pain, and gouty arthritis pain with self-limiting, often within a few days or a few weeks away, many people will "forget the good scar pain", and not to Let it be. In fact, this is wrong. From the orthopedic point of view, the crystallization of uric acid in the body has not disappeared, not treated in time, continuous deposition of uric acid crystals, gouty arthritis will be a recurring problem, a doctor from the endocrine point of view, high serum uric acid did not disappear, it will affect the whole body the various organs, for example, it will lead to metabolic cardiovascular risk factors such as hypertension, hyperlipidemia, Ⅱ diabetes, obesity, insulin resistance associated with and influence each other, and is likely to confirm a direct result of cardiovascular disease.
Hyperuricemia on renal damage: caused by uric acid nephropathy, kidney stones and renal failure. Serum uric acid caused by the risk of renal dysfunction than proteinuria even higher.
Hyperuricemia as a composition of one of the metabolic syndrome, diabetes, obesity, hyperlipidemia and insulin resistance accompanied by simultaneously exist, or reinforce each other, closely related.
Life guidance
Including lifestyle changes and risk factors of control.
Aggressive treatment of elevated serum uric acid associated with cardiovascular and metabolic risk factors. Cardiovascular risk factors and cardiovascular diseases including: hypertension, impaired glucose tolerance or diabetes, hyperlipidemia, coronary heart disease, stroke, heart failure, kidney dysfunction.
Active antihypertensive lipid-lowering therapy; to correct lipid metabolism disorder, improving insulin resistance; and timely control of high uric acid, to protect renal function.
The increased use of drugs and uric acid excretion of uric acid inhibit the synthesis of drugs, lowering uric acid in the treatment goal is to promote crystal dissolution and prevent crystal formation, which need to make a long-term serum uric acid level below the saturation point of monosodium urate; detection of serum uric acid; early detection of High uric acid groups, the control kidney disease, metabolic diseases, cardiovascular diseases and other relevant risk factors, reducing target organ damage.
What people tend to be entangled hyperuricemia
1. There is family history and obesity were
2. Have high blood pressure, high cholesterol, arteriosclerosis, coronary heart disease, diabetes and other diseases are
3. Patients on certain drugs: thiazide diuretics, low-dose aspirin, insulin, glucocorticoid, etc.
4. Trauma and surgery: trauma, burns, surgery, etc.
5. Too greedy people: eating excessive, high-purine diet and alcoholism. We heard that eating seafood, drinking beer can lead to gout, in fact, simply eating seafood, or drinking beer can lead to gout.
Gout patients in daily life
Pipe keep quiet Dodo tired legs do not drink plenty of water
1. Diet to "taboos": low-purine diet and avoid animal offal, seafood. Although the food contained in the pathogenesis of gout purine is not the main reason, but so that the concentration of the blood uric acid increased rapidly, reaching attack critical state. Han Piaoling control foods that are high can reduce the number of acute attack of arthritis.
2. Control of protein intake, less than 1 gram per kilogram of body weight / day.
3. Eat more fresh vegetables, fruits, Eat beans.
4. Avoid alcoholic beverages, including beer and wine. Ethanol contained in wine can increase blood lactate concentration, which can inhibit the renal tubular secretion of uric acid can reduce the excretion of uric acid; the same time, alcohol can make to increase uric acid synthesis. The effects of ethanol on the gout diet is much more serious than that, especially in hunger and eating, after drinking large quantities of high-protein high-purine foods, often can cause an acute attack of gouty arthritis.
5. The water: more than 2000 milliliters per day to ensure that urine output and increase uric acid excretion.
6. The use of oral sodium bicarbonate, urinary pH will be maintained at 6.5-6.9. Alkalization of urine urate crystals in favor of dissolution and from the urine.
7. Be taken to avoid overeating or starvation.
8. Properly handle the triggers, disable or less with the impact of uric acid excretion of drugs, such as penicillin, and high-dose thiazide diuretic drugs such as dapsone, chatter PI, vitamin B1 and B2, insulin and low-dose aspirin.
9. Active treatment and gout-related diseases, such as hypertension, hyperlipidemia, diabetes and coronary heart disease and so on, to prevent overweight. Obesity is necessary to actively lose weight, reduce calorie intake to reduce body weight, sugar accounted for 50% of total calories -60% or less.
10. Gouty arthritis in patients with seizures are often associated with long-distance walking, joint sprains, shoes, discomfort and excessive activity and other factors, which may, after the Department of local tissue injury, caused by uric acid salt off. Thus, gout patients should pay attention to work and rest, avoid over exertion, mental stress, infections, surgery, shoes should be comfortable, without rendering joint injury. Generally not in favor of gout patients in long-distance running or walking tour.